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 Indian J Med Microbiol  
 

Figure 6 (A-M): (A-E) On MRI plaque imaging, plaque appears isointense on PD and hypointense to isointense on T2WI. It is mildly hyperintense on T1WI and isointense on TOF images s/o fibro-fatty plaque. Surface ulceration is present. On MRI, plaque appears isointense on PD (H) hypointense on T2WI (F) mildly hyperintense on T1WI (G) and isointense on TOF (I) suggestive of fibro-fatty plaque. Fibrous cap is seen, but it is discontinuous at places suggestive of surface ulceration. Ultrasound and MRI findings are suggestive of plaque vulnerability. M/67 years with left MCA and MCA-anterior cerebral artery (ACA) watershed territory stroke on diffusion-weighted imaging of the brain (I, J) On CTA, left ICA 80% stenosis. Plaque has attenuation of 37.6 HU (K) Type III ulceration is present (dotted white arrows) (L) On CEUS, plaque neovascularization is seen with surface irregularity (M)

Figure 6 (A-M): (A-E) On MRI plaque imaging, plaque appears isointense on PD and hypointense to isointense on T2WI. It is mildly hyperintense on T1WI and isointense on TOF images s/o fibro-fatty plaque. Surface ulceration is present. On MRI, plaque appears isointense on PD (H) hypointense on T2WI (F) mildly hyperintense on T1WI (G) and isointense on TOF (I) suggestive of fibro-fatty plaque. Fibrous cap is seen, but it is discontinuous at places suggestive of surface ulceration. Ultrasound and MRI findings are suggestive of plaque vulnerability. M/67 years with left MCA and MCA-anterior cerebral artery (ACA) watershed territory stroke on diffusion-weighted imaging of the brain (I, J) On CTA, left ICA 80% stenosis. Plaque has attenuation of 37.6 HU (K) Type III ulceration is present (dotted white arrows) (L) On CEUS, plaque neovascularization is seen with surface irregularity (M)